epileptic attack

Epileptic attack type ( 7 cases ) ;

癫痫发作型7例；

There was an epileptic attack 3 h before examination in the other one case .

另1例于检查前3h癫痫发作1次。

Mechanism of epileptic attack on learning and memory in rats

听源性癫痫发作对大鼠学习记忆的影响及机制探讨

Conclusion The level of GR may associated with epileptic attack .

结论GR的变化可能与癫痫发病有关。

Methods : Classifying 22 cases of EEG in epileptic attack .

达到更好的治疗效果。方法：对22例癫痫临床发作时的EEG进行分析。

No recurrence of epileptic attack was found in 3 patients with epileptic history .

癫痫发作3例术后未再复发；

The Clinical Observation of Calcium and Zinc 's Subsidiary Effect on the Therapy of Epileptic Attack

钙、锌佐治癫痫病发作的临床观察

The frequence of the epileptic attack was another important harmful factor ( P < 0.001 ) .

癫痫本身多次发作也是一个重要影响因素（P＜0.001）。

Conclusion Epileptic attack can be effective controlled by using large dosage of hormone on the basis of routine treatment ;

结论在常规抗囊治疗的基础上大剂量的应用激素能有效的控制癫痫的发作；

Method : 9 cases of this disease were treated with MEBT / MEBO after the epileptic attack had been controlled .

方法：对本组9例患者，在控制癫痫发作下，应用MEBT/MEBO处理烧伤创面。

Results The number of epileptic attack was in direct proportion to concentration of serum creatine kinase and dehydrogenase prehospital .

结果入院前癫痫发作次数与血清肌酸激酶、乳酸脱氢酶浓度呈正相关；

The interrelation between concentration of tetermine in blood , the number of epileptic attack and serum cardiac enzymes .

对血中毒鼠强浓度及癫痫发作次数与血清心肌酶浓度作相关分析。

Purpose : To explore the relations between the course of epileptic attack and the changes of the peripheral microcirculation so as to coordinate the treatment .

目的：为探索癫痫的发病过程与外周微循环改变的关系以便配合治疗。

Epileptic Attack of Tuberous Sclerosis

结节性硬化症的癫痫发作

Results : Before treatment most frequent symptoms were headache , dizziness and epileptic attack ( 82.7 % , 45.1 % and 63.9 % , respectively );

结果：治疗前神经精神症状以头痛、头晕和癫痫发作最为常见（分别占82.7%、45.1%和63.9%）；

Objective To assess the clinical effectiveness of vitamin E adjunctive therapy on intractable epilepsy in children and to approach the relation of epileptic attack and free radical .

目的研究维生素E辅助治疗难治性癫痫的临床疗效，初步探讨癫痫发病与自由基的关系。

Postoperative intracranial hemorrhage was found in 2 patients ( 0.51 % ), 3 patients ( 0.76 % ) had undergone epileptic attack after biopsy . There were no death cases .

并发颅内出血2例（0.51%），癫痫发作3例（0.76%），无死亡病例。

Conclusions The cerebral edema is an important factor of the epileptic attack in early stage while the late epilepsy may be caused by the epileptic focus resulted from the neuroglial scar .

结论脑水肿是早期癫痫发作的重要致痫因素，晚期发作癫痫是因胶质疤痕等因素形成癫痫灶所致。

Epileptic attack is not only temporary brain functional disorder caused by simple alteration of physiological and biochemical , it is important for the changes of spike wave focus form constitution and neurotransmitter to cause epilepsy .

癫痈发作不仅仅是单纯的生理生化改变所引起的暂时性脑功能失调，棘波灶的形态结构和神经递质变化对癫痛的形成和持续状态具有重要作用。

[ Methods ] 23 patients with refractory epilepsy were examined during interphase of epilepsy and epileptic attack by 18F-FDG PET . The results of PET were compared with that of MRI and EEG .

[方法]对23例癫痫患儿于发作期及发作间期行18F-FDGPET检查，并与MRI及EEG比较。

The observation went on 1 year to 3 years and 10 months , average time was 16.2 months . The frequency of epileptic attack 20 weeks and 1 year after treatment was compared with the frequency of basic stage 3 months before treatment .

观察时间为1年~3年10个月，平均16.2个月，治疗20周后及1年后与基础期（入组前3个月）月发作频率进行比较。

Results In the follow up period ( 2-3 years ) was observed heal in 54 cases , among which 6 cases epileptic attack one time a year and 2 cases two time a year , 1 case death , 20 cases operative patients healed .

随访63例（2～3年），治愈54例，有6例1年发作1次，2例1年发作2次，死亡1例。其中手术治疗的22例中，治愈20例。

After praziquantel treatment psychiatric symptoms including headache , dizziness , epileptic attack , nausea , vomiting , neurasthenia , papilledema . depression and dyskinesia were significantly decreased compared with pretreatment ( P0.05 ~ 0.01 ) .

抗囊治疗后，头痛、头晕、癫痫发作、恶心呕吐、神经衰弱症候群、视乳头水肿、抑郁症状、肌体运动障碍等神经精神症状均较抗囊治疗前明显减少（P0.05～0.01）；

Results Observation on the animal behaviors revealed that the extent of epileptic attack was moderate in IL-1 β and IL-6 groups . There was no epileptic attack in control group , GC + IL-1 β group , GC + IL-6 group and GC alone group .

结果动物行为学观察显示IL-1β组、IL-6组癫痫发作程度达中度，GC+IL-1β组、GC+IL-6组和单纯GC组则无明显癫痫发作。

These results have given new evidence that the CVNs might fire in a seizure-like pattern during epileptic attack , which might be responsible for the neurogenic ictal bradyarrhythmia , cardiac asystole , or even the sudden deaths of patients of epilepsy . 3 .

这些结果提供了新的证据，提示癫痫时CVN可能以痫样模式发放。而这种发放模式可能与癫痫病人发作时的神经源性发作性心动过缓、心跳骤停、甚至猝死有关。